Hipotiroidismo primario, insulinorresistencia y la aterosclerosis carotídea subclínica
Abstract
colesterol total, hormona estimulante de la tiroides y grosor íntima-media carotideo fueron significativamente mayores respecto a los insulinorresistentes. El HOMA-IR fue significativamente mayor en el grupo con insulinorresistencia. El valor de TSH ≥ 4,20 µmol/L mostró sensibilidad de 95,5 % y especificidad de 73,3 % en la predicción de aumento del GIMC. El HOMA-IR ≥3,10 tuvo sensibilidad de 95,5 % y especificidad de 73,1 %.
Conclusiones: El hipotiroidismo y la insulinorresistencia son predictores independientes de aterosclerosis carotídea subclínica.
Palabras clave: hipotiroidismo; insulinorresistencia; grosor íntima-media carotideo.
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